Klionsky, D., Abdelmohsen, K., Abe, A., Abedin, M., Abeliovich, H., Arozena, A., … Zughaier, S. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition). Autophagy, 12(1), 1-222. https://doi.org/10.1080/15548627.2015.1100356 (Original work published 2016)

Guo, Z., Ersoz, A., Butterfield, D., & Mattson, M. (2000). Beneficial effects of dietary restriction on cerebral cortical synaptic terminals: preservation of glucose and glutamate transport and mitochondrial function after exposure to amyloid beta-peptide, iron, and 3-nitropropionic acid. Journal of Neurochemistry, 75(1), 314-20. https://doi.org/10.1046/j.1471-4159.2000.0750314.x

Begley, J., Butterfield, D., Keller, J., Koppal, T., Drake, J., & Mattson, M. (1998). Cryopreservation of rat cortical synaptosomes and analysis of glucose and glutamate transporter activities, and mitochondrial function. Brain Research. Brain Research Protocols, 3(1), 76-82. https://doi.org/10.1016/s1385-299x(98)00024-5

Bruce-Keller, A., Begley, J., Fu, W., Butterfield, D., Bredesen, D., Hutchins, J., … Mattson, M. (1998). Bcl-2 protects isolated plasma and mitochondrial membranes against lipid peroxidation induced by hydrogen peroxide and amyloid beta-peptide. Journal of Neurochemistry, 70(1), 31-9. https://doi.org/10.1046/j.1471-4159.1998.70010031.x

Subramaniam, R., Roediger, F., Jordan, B., Mattson, M., Keller, J., Waeg, G., & Butterfield, D. (1997). The lipid peroxidation product, 4-hydroxy-2-trans-nonenal, alters the conformation of cortical synaptosomal membrane proteins. Journal of Neurochemistry, 69(3), 1161-9. https://doi.org/10.1046/j.1471-4159.1997.69031161.x

Hensley, K., Butterfield, D., Hall, N., Cole, P., Subramaniam, R., Mark, R., … Aksenov, M. (1996). Reactive oxygen species as causal agents in the neurotoxicity of the Alzheimer’s disease-associated amyloid beta peptide. Annals of the New York Academy of Sciences, 786, 120-34. https://doi.org/10.1111/j.1749-6632.1996.tb39057.x (Original work published 1996)

Mark, R., Hensley, K., Butterfield, D., & Mattson, M. (1995). Amyloid beta-peptide impairs ion-motive ATPase activities: evidence for a role in loss of neuronal Ca2+ homeostasis and cell death. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience, 15(9), 6239-49. Retrieved from http://www.jneurosci.org/cgi/pmidlookup?view=long&pmid=7666206

Hensley, K., Carney, J., Mattson, M., Aksenova, M., Harris, M., Wu, J., … Butterfield, D. (1994). A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease. Proceedings of the National Academy of Sciences of the United States of America, 91(8), 3270-4. https://doi.org/10.1073/pnas.91.8.3270 (Original work published 1994)

Mattson, M., Lovell, M., Ehmann, W., & Markesbery, W. (1993). Comparison of the effects of elevated intracellular aluminum and calcium levels on neuronal survival and tau immunoreactivity. Brain Research, 602(1), 21-31. https://doi.org/10.1016/0006-8993(93)90236-g (Original work published 1993)

Mattson, M., Lovell, M., Furukawa, K., & Markesbery, W. (1995). Neurotrophic factors attenuate glutamate-induced accumulation of peroxides, elevation of intracellular Ca2+ concentration, and neurotoxicity and increase antioxidant enzyme activities in hippocampal neurons. Journal of Neurochemistry, 65(4), 1740-51. https://doi.org/10.1046/j.1471-4159.1995.65041740.x